Quiz 3 Notes
Quiz 3 Notes PSY 3150 SEC001
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This 2 page Bundle was uploaded by Jefferson Oakey on Monday February 22, 2016. The Bundle belongs to PSY 3150 SEC001 at University of Colorado Colorado Springs taught by Robert L. Durham in Spring 2016. Since its upload, it has received 32 views. For similar materials see Psychology of Motivation in Psychlogy at University of Colorado Colorado Springs.
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Date Created: 02/22/16
Physiology and Hunger Wednesday, February 10, 2016 10:51 AM Stimulate Ablate Operant Conditioning More Eating No Eating • BF Skinner ○ A Theoretical - Psychology is not ready for theories ○ Requires description Excitatory Homeostasis • Hypothalamus ○ Changes in blood chemistry and glucose Glucose ○ Maintains a temperature and respiration rate Less Eating Constant eating ○ Optimal level of eating behavior Hormones • Glucoreceptors Inhibitory ○ Short-term regulation - monitoring the internal environment Lot of glucose, don't need to eat Lack of glucose, search for food Hypothalamus □ Ventromedial Lesions - Hyperphagia Satiety center - turns off eating when sufficient □ Lateral Lesions - stop eating aphagia Hunger center (excitatory - turns on eating) ○ Long-term regulation Eat everything we could during good times to prepare for the bad times □ May be a reason why we have obesity problem □ Evolution There are systems (lipostatic theory) that monitor body fat levels homeostatically (Set-Point theory) □ We have a normal body weight and the LH regulates that weight (Set Point) □ Damaged LH, when recovered, rats have a new, lower, set point, (VMH plays a role too) □ Remove fat, animal regenerates to previous level Theory is that the hypothalamus regulates this, fat levels in the body □ The hormone, Leptin, produced by fat cells indicates how much fat is available Lean people or rats have lower leptin levels Leptin levels detected by arcuate nucleus in hypothalamus □ Weight loss stimulates leptin and insulin levels drop and arcuate nucleus secretes neurotransmitters (neuropeptide y) resulting in food intake and weight gain □ Other neurotransmitters are involved ○ There are back up centers and multiple cues to start/stop eating ○ Hormone Ghrelin Glucoreceptors in duodenum Liver, Pancreas, Stomach Not sure how they interact with each other Quiz 3 Page 1 Arousal and Stimulus Wednesday, February 17, 2016 10:43 AM Excitatory Questions • Neurotransmitters either stimulate and inhibit? ○ Serotonin stimulates ○ Dopamine inhibits Neuron firing ○ A neurotransmitter can do one or the other It can't do both ○ Nerve cells are always firing You will see some level of activity If excitatory event occurs then you see a spike in activity If you see an inhibitory event, spike goes down Inhibitory Stimulus • Stimulus response argument - Hull ○ Very open to criticism ○ Thought is not a part of this Stimulus External Drive ○ Action without thought ○ The more reinforced trials the more sHr ○ The more drive condition the faster and the more the learning ○ The stronger the stimulus, the more and faster the learning ○ The more reward, the more learning - if some is good more is better Reaction Amplitude (Arousal) ○ The amount of work coupled with the level and accessibility of reward ○ If sEr net = (sHr x D x V x K) - sIr > sLr, then R Maybe not multiplicative but additive Hull's model is a lot like Freud's model □ Negative motivational state that you seek to reduce □ Drive reduction theory • Spence Stimulus Internal ○ Maybe not seeking to reduce drive, but looking for an optimal level of stimulation Based on homeostasis Response Habit Family Hierarchy • Rats • sUr - stimulus unlearned response ○ Hooded rats - smart Electrify a grid that the rat is on, the rat will learn in one trial to move • sHr - stimulus habit ○ Holtzman rats - not as smart response Electrify grid, the rat will lift a foot or defecate Quiz 3 Page 2
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