Exam 1 study guide
Exam 1 study guide PSYCH101
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This 5 page Study Guide was uploaded by Valerie Ho on Tuesday February 3, 2015. The Study Guide belongs to PSYCH101 at University of Washington taught by Dr. Lauren Graham in Fall2014. Since its upload, it has received 257 views. For similar materials see Introduction to Psychology in Psychlogy at University of Washington.
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Date Created: 02/03/15
Study Guide for Exam 1 Final New content to Final version in red bold font L content was only in lecture T content is only in your textbook Lectures 15 16 and Chapter 1 Neuroscience T Biological psychology T Persistent issues in biopsychology L Mind versus brain L but see overlap on page 13 consciousness section Descartes Dualism Localization of function Phrenology Neuroplasticity Neural communication L but see overlap on page 9 point 4 Santiago Ramon y Cajal L quotNeural Doctrinequot L Somatic intervention T Independent variable T Dependent variable T Behavioral intervention T Correlation T Causality T Lectures 17 112 and Chapter 2 Neurons 80bi11ion Glial cells Relative and approximate numbers of neurons amp glia in brain L Golgi stain Nissl stain Dendrites Cell bodySoma Axon Axon hillock Axon terminals Sensory neurons T Motor neurons T Interneurons T Multipolar neurons Bipolar neurons Unipolar neurons Presynaptic membrane Postsynaptic membrane Synaptic cleft Synapse Synaptic vesicles Neurotransmitter Receptors Oligodendrocytes Schwann cells Myelin Multiple sclerosis T Nodes of Ranvier Astrocytes Microglia Central nervous system T Peripheral nervous system T N0T cranial or spinal nerves Nerves versus tracts Ganglia versus nuclei Autonomic nervous system T Sympathetic versus parasympathetic nervous systems T ONLY to the extent of the three text boxes in Figure 29 page 33 Cerebral cortex Gyrigyrus versus sulcisulcus Four lobes of cortex Corpus callosum The main band of axons that connects the two hemisphere White matter vs gray matter Layers of cerebral cortex Figure 213 p38 Skullleeningesleortex Meninges l Dura mater 2 Arachnoid 3 Pia mater CSF in between arachnoid and the pia mater Collection of regions in the forebrain Movement Amygdala Hippocampus Thalamus Hypothalamus Midbrain Manages survival functions Maj or source of dopamine Substantia nigra Substantia Nigra Part of the midbrain but also crosses Basal Ganglia Source of dopamine Parkinson s Disease L Cerebellum Meninges Cerebrospinal uid Lateral 0 All related to the CSF and coverage of the brain Bloodbrain barrier Lectures 113 115 and Chapter 3 Polarization Ions Resting potential Ion channel Potassium ions K Diffusion Concentration Diffusion gradient L Electrostatic pressure Electrical gradient Sodiumpotassium pumps Sodium ions Na Equilibrium potential 0 K potential 60mV 0 Na potential 40mV Hyperpolarization Depolarization Threshold 40mV 0 Na ooding into the axon Action potential detailed steps e g Figure 36 and lecture on ll4 0 Threshold leads AP 0 A brief reversal of a neuron s membrane potential Allornone property T Voltage gated Na channel Voltage gated K channel Refractory phase T Saltatory conduction Multiple sclerosis T Neurotransmitter Presynaptic vs postsynaptic cells Postsynaptic potential PSP EPSP IPSP Chloride ions Cl 0 Causes the neuron to be hyperpolarizes 0 Due to the opening of channels by ACh during inhibitory synapses Spatial summation Temporal summation Synaptic transmission Synaptic vesicles Synaptic cleft aka synaptic gap Calcium ions Ca2 Ligand Receptors Agonist Antagonist Degradation Reuptake Transporters Types of synaptic connections T see figure 313 0 Axodendritic axosomatic axoaxonic dendrodenritic Epilepsy T 0 Causes seizures 0 Disorder marked by sudden loss of consciousness associated with abnormal electrical activity in the brain Seizures T Caused by excess glutamate too much AP and neurons firing Mapping the human brain T Figure 317 Lectures 120 122 and Chapter 4 Exogenous T Endogenous T Ionotropic receptor T Metabotropic receptor T Discovery of acetylcholine see Fig 43 0 Otto Loewi and frog hearts 0 Heart rate slowing Acetylcholine Basal forebrain 0 Major source for ACh Dopamine 0 Produced in the substantia nigra with is in the midbrain Ventral tegmental area Note substantia nigra is in Ch 2 list 0 In the midbrain projects dopamine to the nucleus accumbens Glutamate GABA Ligand Metabolic tolerance T Functional tolerance T Downregulate T Upregulate T Drug effects on transmitter production Always decreases the production of NTs transmitter release Blocking NT transmissions and competes of receptors transmitter clearance everything that interferes with the 3 processes of NT elimination receptor activity Blocks NT from receptor activates the receptor like normal NT postsynaptic activity Blocks the release of enzyme to open ion channels Note for above terms understand general mechanisms of how drugs can affect each stage and how a drug could be an agonist or antagonist at each stage That is learn general mechanisms but not specific drug examples in Figure 47 and 48 Neuroleptics Antipsychotics Monoamine oxidase MAO inhibitors Selective serotonin reuptake inhibitors SSRIs Benzodiazepines Morphine Heroin Endogenous opioids T Nicotine Alcohol THC Endocannabinoids T Caffeine Cocaine Amphetamine LSD MDMA Tell Tale Brain There will be questions on the exam that relate to the assigned chapters from VS Ramachandran s The Tell Tale Brain I expect you to have read the chapters understood them and thought about them I won t try to trick you if I ask questions about these chapters but I will at least include questions that test whether you did the readings Finally you will spend some time in each Friday quiz section discussing the week s chapter Test questions may be inspired by these discussions Assigned chapters see additional notes in the TT Brain Reading Schedule on the course website Introduction No Mere Ape Chapter 1 Phantom Limbs and Plastic Brains Chapter 2 Seeing and Knowing
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