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Myasthenia Gravis Study Guide

by: Alexis Walton

Myasthenia Gravis Study Guide Bio 308

Marketplace > Winthrop University > Biology > Bio 308 > Myasthenia Gravis Study Guide
Alexis Walton

GPA 3.0

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About this Document

This is a study guide on myasthenia gravis made from watching the video online uploaded by Dr. Guenther
Human Physiology
Dr. Guenther
Study Guide
Myasthenia, Gravis, Human, Physiology
50 ?




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This 2 page Study Guide was uploaded by Alexis Walton on Friday September 16, 2016. The Study Guide belongs to Bio 308 at Winthrop University taught by Dr. Guenther in Fall 2016. Since its upload, it has received 57 views. For similar materials see Human Physiology in Biology at Winthrop University.


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Date Created: 09/16/16
Myasthenia Gravis Study Guide (Over View of video and PowerPoint) Neuromuscular junction- contact being made on the axon of a motor neuron and the muscle (skeletal muscle) the axon has synaptic vesicles and it releases Ach. The motor end plate is where Ach binds. Synaptic Junction Vesicles with ACh- Axon contains vesicles with Acetylcholine (Neurotransmitter) Muscle- The skeletal muscle has the ACh receptors. More specifically the Nicotinic Acetylcholine Receptors. Normal Nerve Impulse Transaction. 1. Receive nerve impulse all the way to the end of the axon. 2. Then release the neurotransmitter. 3. ACh would then come over and bind to its receptors on the muscle @ the motor end plate. 4. The binding of ACh on the motor end plate initiates muscle action potential and muscle contraction. Myasthenia Gravis is thought to be an autoimmune disorder, the body is attacking itself in a sense. Myasthenia Gravis Nerve impulse transaction. 1. Body generates antibodies that target the Acetylcholine receptors. a. It is actually thought that it reduces the number of receptors so that there are very few left b. ACh will not be able to bind to it’s receptor. c. In turn there would be not muscle Impulse and no muscle contraction. d. Thus creates weakened muscles. 2. Characteristics a. Weakened muscles b. Drooping eyelids (Ptosis) Potential Treatment for Myasthenia Gravis 1. Acetylcholinesterase- an enzyme that would target ACh and break it down. 2. Drug created to prevent Acetylcholinesterase which is Neostigmine which should create and increased amount of Acetylcholine in the synaptic cleft. 3. Increased ACh increases the chance for ACh to bind to any of the ACh receptors that are left 4. Which hopefully improve the ability to generate the muscle impulse. 5. Therefore, improve muscle contraction for patients There is more than one type of treatment drug for myasthenia gravis!!


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