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CSU - ANEQ 346 - Study Guide for Exam 3 ANEQ 346 - Study Guide

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CSU - ANEQ 346 - Study Guide for Exam 3 ANEQ 346 - Study Guide

School: Colorado State University
Department: Science
Course: Equine Disease
Professor: Hess
Term: Summer 2016
Tags:
Name: Study Guide for Exam 3 ANEQ 346
Description: Study Guide for Exam 3
Uploaded: 11/13/2016
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background image Exam 3 Study Guide for ANEQ 346 1. What is the percentage of race horses that are affected by tendon diseases?
2. What is the difference between a tendon and a ligament?
3. List the 3 phases of tendon injuries:
4. How does the chronic phase heal?
5. What does the tendon structure support?
6. How should you manage the sub acute phase?
7. Why is platelet rich plasma effective in treating tendon injuries?
8. Who has a higher chance of getting tendonitis, a race horse or a show 
jumper? 9. What is the most mobile joint?
10.Is lymphangitis bilateral or unilateral usually?
11.List 3 reasons that Bog Spavin can result from:
12.Define Thoroughpin:
13.What is consolidation?
14.What tends to cause more problems in horses? O2 movement or CO2 
movement? 15.When a horse has a stertor noise what does it sound like?
16.What is the correct term to describe a horses normal breathing sound?
17.Why do people use the rebreathing exam?
18.Where do rattle snake bites commonly occur on the horse?
19.What is a treatment option for laryngeal dysfunction?
20.What age horses does pharyngeal lymphoid hyperplasia commonly affect?
21.Name the bacteria responsible for strangles:
22.How is strangles transmitted?
23.How long is a horse considered contagious if it has been diagnosed with 
strangles? 24.What does EIPH stand for?
25.What is pleuropneumonia?
26.Does equine influenza virus have any secondary viral or bacterial infections?
27.List some ways a horse can obtain equine herpes virus:
28.How often do you vaccinate for equine herpes virus?
29.Who is rhodococcus pneumonia most likely to affect?
30.What are two common allergic diseases we went over in lecture?
31.Who is infectious diseases most common in?
32.What is the most commonly used infection control?
33.Why aren’t vaccines enough when it comes to controlling infection?
34.List some common ways to prevent infectious diseases:
35.What are some ways as a horse owner to manage exposure when it comes to 
your barn? 36.What are arguably the two most important key factors when it comes to  preventing infectious dieases?
background image Key: 1. 40%
2. Ligaments connect bone to bone and tendons connect muscle to bone
3. Acute phase, Sub Acute phase. Chronic phase
4. By cross sectioning instead of running parallel
5. Metacarpal phalangeal joint
6. Doing walking exercises with ultrasound monitoring, intra-lesional therapy
7. It increases blood flow
8. Race horse
9. Tibiotarsal (Tarsal-curual) joint
10.Unilateral
11.Bad shoeing, poor conformation, OCD
12.Distention of tarsal sheath of the deep digital flexor tendon just above the 
hock 13.Collapse of the lower airway
14.O2
15.Snore
16.Bronchvesicular sound
17.It increases CO2
18.Nose
19.Tie back
20.1-3 year olds
21.Gram positive beta-hemolytic bacteria
22.Direct contact of discharge or exposure to fomites
23.6 weeks
24.Exercise induced pulmonary hemorrhage
25.Mixture of pleuritis and pneumonia
26.Yes, bacterial infection
27.Sharing a water sources, training facilities, nose to nose contact, shows and 
events 28.Every 6 months
29.Foals ages 1-4 months
30.Inflammatory airway disease and recurrent airway obstruction
31.Young and old horses
32.Vaccines
33.Some diseases don’t have a vaccine
34.Minimize transport stress, provide clean water, insect and rodent control, etc
35.Have visiting horses meet health requirements, take precautions with horses 
returning from events, isolation of new additions 36.Basic hygiene and pre-planning Study Guide Other Ways to Optimize Resistance beyond Vaccination: what 
are important measures?
Minimize transport stress, optimize ventilation, temperature 
control, good nutrition and clean water, insect and rodent 
control
a Options for Management of Exposure Risk: what are risks? That it will come at a cost, not as easy as vaccinations
background image b Options for Managing Exposure Risk Posed by Horse Contact Health requirements for visiting and new horses, precautions 
with horses returning from events, segregation of horses in 
groups by risk level, isolation and monitoring of new additions, 
monitoring for signs of disease
c Example of Health Requirements Certificate of veterinary inspection, past medical history, 
examine horse once it arrives, test for infection
Respiratory Diseases Dr. Swain What is the function of the sinuses? Produce mucus that moistens the inside of the nose, 
also lightens the skull
What are the main parts of the larynx? Arytenoid cartilage, opening to trachea, vocal cord, 
epiglottis
What are the structures in the:
 guttural pouch –
 
cranial nerves, sympathetic trunk,  external and internal carotid arteries. Covers the 
temporohyoid joint. Located behind the cranial cavity, 
lies between the pharynx and beginning of 
oesophagus.
 stilohyoid bone –  guttural pouch, cranial nerves trachea -   begins at the larynx and ends at the hilus  where it divides into the two bronchi carina –  ridge of cartilage in the trachea between the  division of the two main bronchi  bronchi –  enter the lung What is the specific characteristic of the 
epithelium? What is its function?
Made up of cilia, goblet cells and mucus, Lines the 
organs, vessels and body cavities
Exposed to fluid and air. Cells in the epithelium are closely packed together 
to help it form a protective barrier. Protects inner 
structures from dehydration and damage.
What is the mediastinum? What passes Lining separating pleural space from other thoracic 
organ structures. Blood vessels pass through.
Alveolar structure and surfactant Structure is called an alveolus. It contains alveolar 
type I and type II cells and inner surfactant layer. Also 
contains lamella body, tubular myelin, alveolar fluid 
and alveolar macrophage.
Lung injury and structures
background image Consolidation = collapse of lower airway
Can also have abscess formation which can lead to 
airway damage and inflammation.
Decreased gas exchange surface area
Contains the bronchi and alveolus, fibrin tissue
Nasal flow Should be equal. If reduced, there is some obstruction.
Also look for nostril flare
Types of nasal secretion Discharge either being serous or mucoid. Different 
color also. Blood would mean epistaxis.
Cough, respiratory effort Sign of exercise intolerance. Another name for 
respiratory rate and effort is tachypnea. Phase of 
respiration should be slow inspiration and expiration
Respiratory noise Stridor, stertor, snore, wheeze,  Fever Sign of respiratory infection Auscultation noises Normal = bronchovesicular sounds
Abnormal = crackles, wheezes, tracheal rattle
Rebreathing exam function Horse breathes into bag. Increased CO2 and 
encourages deeper breaths. Asses tolerance which is 
seen by coughing, rapid breathing and prolonged 
recovery
Testing for respiratory disease: viral testing and 
bacteriological disease
Cultures, PCR and virus isolation Ultra sound and x-ray Thoracic ultrasound – should clip to make it easier, 
evaluates the pleural surface and pleural fluid 
presence
Transtracheal wash Culture and cytologic analysis Endoscopy Helps to visualize masses, determine if it’s a 
congenital or acquired defect and is able to asses 
during exercise
Broncoalveloar wash Cytologic analysis Upper airway:
background image Rattlesnake bike, signs and what you can/should do. Happens on the nose most often, can lead to edema and respiratory 
distress, can perform emergency tracheostomy to help
Ethmoid hematomas : clinical signs, what can be done Not a tumor but a low grade unilateral epistaxis. Can be treated by 
injecting formalin, a surgical resection. Reoccurrence is common
Nasal polyps: signs, occurrence Obstruct airflow. Result from chronic irritation Sinusitis: primary and secondary signs and treatment Unilateral discharge. Primary includes cushings syndrome. Secondary 
includes bad teeth and smell
Roaring horse with respiratory stertor: laryngeal hemiplegia: why 
does it happen?
Roaring = noise that happens during exercise and sounds like a snore. 
Happens to horses ages 3-7. Happens due to nerve injury – branch of 
vagus nerve. Tie back is the most common treatment. Risks include 
aspiration pneumonia and failure of technique.
Arytenoid chondritis: what is it? Exercise intolerance and respiratory noises. Common in race horses Epiglotis entrapment: what happens? Membrane covers the epiglottis called the aryepiglottic fold.  Strangles: clinical signs, transmission, passive carriers, bastard 
strangles, treatment
CONTAGIOUS! Signs include abscess formation. Transmission is from horse
to horse, direct contact with discharge. Horses considered contagious up 
to 6 weeks. Bastard strangles is a metastatic spread of abscesses. 
Treatments include vaccines and antibiotics
Guttural pouch mycosis: signs, consequences Signs include difficulty swallowing and nose bleeds. Consequences include
cranial nerve damage
EIPH: Clinical signs, cause, types of horses affected, treatment 
and management
Signs include exercise intolerance and on rare occasions, bloody noses. 
Happens in high performance horses. Treatments and managements 
include nasal dilator bands and furosemide.
Shipping fever: reasons, predisposing factors Transport stress. Predisposing factors include strangles ­ Influenza >> Horses unique: Pleuritis + Pneumonia = 
“Pleuropneumonia” 
o Pleuropneumonia has three Stages:  o I  Exudative stage, 

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School: Colorado State University
Department: Science
Course: Equine Disease
Professor: Hess
Term: Summer 2016
Tags:
Name: Study Guide for Exam 3 ANEQ 346
Description: Study Guide for Exam 3
Uploaded: 11/13/2016
30 Pages 116 Views 92 Unlocks
  • Better Grades Guarantee
  • 24/7 Homework help
  • Notes, Study Guides, Flashcards + More!
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