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OSU - NEUROSC 3010 - Neuro 3010 Exam 2 - Study Guide

Created by: Tori Carney Elite Notetaker

Schools > Ohio State University > Neuroscience > NEUROSC 3010 > OSU - NEUROSC 3010 - Neuro 3010 Exam 2 - Study Guide

OSU - NEUROSC 3010 - Neuro 3010 Exam 2 - Study Guide

School: Ohio State University
Department: Neuroscience
Course: Introduction to Neurophysiology
Professor: Georgia Candice Askwith
Term: Fall 2015
Tags: neurophysiology
Name: Neuro 3010 Exam 2
Description: This is the first half of the study guide! The rest will be up tomorrow! Enjoy! Good luck!
Uploaded: 09/28/2017
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School: Ohio State University
Department: Neuroscience
Course: Introduction to Neurophysiology
Professor: Georgia Candice Askwith
Term: Fall 2015
Tags: neurophysiology
Name: Neuro 3010 Exam 2
Description: This is the first half of the study guide! The rest will be up tomorrow! Enjoy! Good luck!
Uploaded: 09/28/2017
8 Pages 218 Views 174 Unlocks
  • Better Grades Guarantee
  • 24/7 Homework help
  • Notes, Study Guides, Flashcards + More!

Unformatted text preview: LCIUNT 1 3010 Exam 2 Purkinje (cil - many branches -in.artbellum - 1 climbing fiber per al (complex spire) Signals along branches parajke fiber in out - goes to purkinje Cimpu spike) using TIX: - DWOKS soma spikes but not dindine spices - cat is usd in mu spikes no.Cat = no activanon P-type T-type are responsibu - w-agatoxin + 2 is antagonist IS antagonist complex spike - glutamat ruase onto PimPA MGIURS On pucoc Soma 3 dundries - glu = excitatory - Naf = soma spine conductances - many dendrite synapses TNT reuase Inaiu a widespread depolari anon Tat (oods To Threshoid maunad for vortovi gaid cart channes (PT) - Catt inf lux a ktinflux for fast repolarijanun between spikes and to 6100 DUP following spites_ NAT cat AHP>ata CRH (corticoid-roulasing-homeru CRF jocurebelum - normally stimulates Glu - antagonist = JAHP is blocked -TAHP) > C&Fs Suppress PHP (dose dependent) CRE may be suppressing Ka channels - hy per polari catun can atter_Spike Train proven bu when -75mVdlupolarrood mu onset of neis delayed 1 amplitude of depolariting signal = Tfiring rare - What is significanu of firing rate? NSIS frequency modulated __ amplitude of op doesn't a significantly * Ifrio = TNT - Tognal to post synapnc_ into on intensity" - ca uvels tage determine firing 0 -55 m = ca channels inaan ve __ depolantation- AP Tran . - (OmyCa' channels inactive dipolari Won = no response (Ap) -70 mV ca channels active men inactive influx = SI0w wave depolaritation L>Short burst of op some neuroos mandomly fire - influx of ca_when hy perpolariud - KOER VgNa open A'P_ - ca inactivat when depolarized - HOW 0ftch do neurins fire? 200-300 Spikes sec (Hz) - What determines? abs. refractory : 1-2 MAL Irelan ve (etractory = 2-5 msec Threshold retury to baseinu in 3-5 msecRest vg Na Q QS Inacni ve opon /active Refractory periods - Apsolutt. (cil is incapable of responding to dupolaritanol rtgauraless of intensity InactivatdVg Nat Channels opening of vo Kt channels - relativt Ketractory . ceul_iscapuduo of op only if stimullisis high, Som'Ng Nai art ready. Rest Vg kt channes are closing - duration of absolut refractory = cycles 1 cyul 1000 ms. 1000 cycus 1000 Hz 1 MS. 1SS - HOW to measure refractory periods Abs generate ne at same (urel of Stimuui A. de termine when you can mecord another AP from a se coser to initial segment sus progressively longer INANVAS prwn. Simuu (IST) prop. AP ISI Relative; -generate pes 'us (orger intervals (ISI) eac n hme demiru when you can record oundnar Are

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