study guide exam 2 (part 3)
study guide exam 2 (part 3) PSYC 2015
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This 1 page Study Guide was uploaded by Jennifer Gittleman on Wednesday October 28, 2015. The Study Guide belongs to PSYC 2015 at George Washington University taught by Dr. Wu in Fall. Since its upload, it has received 39 views. For similar materials see biological psychology in Psychlogy at George Washington University.
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Date Created: 10/28/15
Digestion begins in mouth where enzymes in saliva break down carbs hydrochloric acid amp enzymes Carbohydrates pepsin in stomach digest proteins small intestine 5am has enzymes that digest proteins fats amp carbs amp absorb Mesh bloodstream large intestine absorbs water amp minerals 8 pass as feces Regulation of Feeding stomach amp duodenum me stop eating is the distention of the stomach vagus I Abmrb digested nerveconveys info about stretching of stomach walls 39 39 39 39 teenage to brain splanchnic nerveconveys info about nutrient contents of the stomach cholecystokininCCKreleased amp regulates hunger by closing sphincter muscle bw sto duodenum amp cause stomach to hold contents ll faster nerve to send message to hypothalamus that releases c cmllillr39 Regulation of Feeding glucose insulin amp 7 quot E glucagon in uence feelings of hunger most mm ate 39 himjmnh 39 digested foods enter bloodstream as glucose source mineral l 7 quot mmmm cafl gl liarquot 39 Ellie l til immune 5 r air of energy Explanation of glucagoninsulin pic when glucose ll convert some of excess into glycogen amp fat cells convert converts glycogen back into glucose insulin pancreatic hormone that enables glucose to enter cell insulin levels rise as someone is getting ready for a meal amp after a meal high insulin level gain weight Dglucagon hormone released by pancreas when glucose levels fall stimulates the liver to convert some of its stored glycogen to glucose to replenish low supplies in blood Type I Diabetes insulindependent 1 Stomach changes food into glucose 2 Glucose enters bloodstream 3 Pancreas malltes littleno insulin 4 Littleno insulin enters bloodstream 5 Glucose builds up in bloodstream Type II Diabetes adultonset normal process glucose absorbed insulin resistance glucose stays in blood Leptin longterm regulation of feeding signals the brain to increase or decrease eating Obesity caused by consistent overeating damages endoplasmic reticulum in neurons of hypothalamus leads to decreased leptin sensitivity people with a mutated gene for the receptors melanocortinneuropeptide responsible for limiting food intake overeat amp become obese Prader Willi syndrome is a genetic condition marked by mental retardation short stature and obesity blood levels of ghrelin are 5 times higher than normal S mmli39h s form 311 W Elysee lt Stimulates glueoe uptake Two kinds of neurons Axons from satiety mm mm in the arcuate nucleus sensitive cells of arcuate of the hypothalamus nucleus deliver an Mum mlquot excitatory message to paraventricular nucleus which triggers satiety rennin kidney fat 7 ii NW Ang E GABA melanocortin chemical RGd important for limiting food lwikllnds olf neulrons in h i bit I e 39 in t e atera nuc eus 39 HILIl gE llii Intake39 NPYAgRP bIOCk of the hypothalamus satiety action of motive paraventricular nucleus amp Ghrelinr Dlr39exinfhypocretih e Nucleus increases arousal MEla DCDHin NLEUIDns paraventricular nucleus Neurons in the Effects of H othalami paraventricular nucleus ml WE hyp thalamus yp of the hypothalamus V I Saltietymotive l Output to other areas including Hypothalamic Area EHECt M Lee cerebral cortex and brainstern Output increases feeding Preoptic area De cit in phys Lateral preoptic area De cit in osmotic th 0 rexi n in C rea ses a i ma 395 I n ofpassing axons Lateral hypothalamus Undereating weight p e rs I Ste n ce n see Kl n 9 food underarousal undemsponsiwms responds to Incentives amp Ventromedial hypothalamus Increased meal frequ rel n f0 rc e m e n t Paraventricular nucleus Increased meal siZe especially increased carbohydrate intake during the rst meal of the active period of the day CC Cenoaoe Learninn 2013
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