Patho I Pre-Exam Notes
Patho I Pre-Exam Notes NURS 239 001
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NURS 239 001
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This 3 page Class Notes was uploaded by Haley on Tuesday February 2, 2016. The Class Notes belongs to NURS 239 001 at University of New Mexico taught by Drexler in Fall 2015. Since its upload, it has received 21 views. For similar materials see Pathophysiology I in Nursing and Health Sciences at University of New Mexico.
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Date Created: 02/02/16
Patho I PreExam Notes Manifestations of Inflammation Local: effects area of injury Systemic: effects entire body heat fatigue, lethargy pain fever, headache edema: swelling weight loss incapacitation: body part is not increased circulation functional increased leukocytes and plasma proteins in blood How to treat inflammation: ● RICE→ reduce blood flow and swelling ● Anti‐histamines→ block chemical mediators Why should we treat it? ● More tissue damage can occur past the area of immediate damage. ● The less time the body is under inflammation, the better. Chronic Inflammation ● Lasts 2+ weeks ● Occurs without acute inflammatory process ● Granuloma: the body walls off damaged tissue ○ Swollen knuckles Tissue Healing ● Inflammatory phase ○ Wound area is cleaned ○ Fibrin mesh is laid down ● Proliferative phase ○ Collagen mesh ○ Scar tissue ○ Cell differentiation begins ● Remodeling phase ○ Fibroblasts complete scar ○ Cells finish differentiating ● Primary intention: wound is closed with sutures or adhesion process ● Secondary intention: wound heals naturally ○ Bowl shape from the bottom up ● Angiogenesis: new blood vessel creation Complications of Tissue Healing ● Ulceration: cells don’t have anything to build next to so they erode themselves ● Infection: a bacteria lodges itself in the wound while it is healing. Then, it multiplies and breaks the wound open again so the process must start over. ● Dehiscence: Wound opens due to internal pressure ○ Abdominal sutures burst when patient uses abs ● Keloids: Overproduction of scar tissue ● Adhesions: stalactite/mite formations that attach to walls to heal Gastritis ● Type of acute inflammation ● Not getting enough good blood flow to the stomach ● Endothelial lining of stomach is damaged because HCl erodes tissue ○ acid+ acid= gastric irritation ● H. pylori: bacteria that eats through tissue ● Symptoms: ○ Relux: undergoes hyperplasia ○ Nausea ○ Vomiting ○ Loss of appetite ○ Pain ● Diagnostic ○ Endoscope ○ Stool Sample ○ What color is poop/vomit? ○ What did they eat? ○ Blood count to test for anemia ● Treatment ○ Buffer acid with base→ Tums, Ca based medication ○ Get rid of substance causing irritation Crohn's Ulcerative Colitis Skip lesions Continuous lesions Small intestine Large intestine (colon) Colon pain Sigmoid colon and rectum Camera pill Crampy pain MRI/CT scan Colonoscopy Smoking cessation Nutritional consult Pinkish ring in toilet water Take anti‐inflammatories Test for occult blood Blood stools Fever, weight loss, fatigue Endemic: disease or condition that is regularly found in an area Pandemic: worldwide or nationwide disease Epidemic: disease in a community at a certain time
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