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Bill 207

by: Tiana Molitor

Bill 207 Biology 207

Tiana Molitor
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About this Document

Week 1 Notes 2-2-2016 -> 2-4-2016
Genetics Ecology Evolution
Leah Domine
Class Notes




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This 4 page Class Notes was uploaded by Tiana Molitor on Friday February 5, 2016. The Class Notes belongs to Biology 207 at University of St. Thomas taught by Leah Domine in Spring 2016. Since its upload, it has received 76 views. For similar materials see Genetics Ecology Evolution in Biology at University of St. Thomas.


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Date Created: 02/05/16
Bio 207 Week 1 Notes 2-2-2016 * Slide 3 - all cells are diverse - proper function of cells is essential to ALL life * Slide 4 -cells don’t live forever - some last longer than others - exposure is a big reason why some cells get replaces more than others ex) skin cells get replaces way more than liver cells - Out of control cell division leads to cancer - Mitosis-> growth, repair, and cell replacement (KNOW) Cell -> Nucleous -> Chomosomes -> DNA (biggest -> smallest) *Slide 7 - cells use DNA to know how to function - Every cell has DNA in the nucleous -DNA replicated perfectly *Slide 8 -46 total, differs with every species - paired based on similar length & coded for what - 23 pair = determines sex of human ( xx- female, xy- male) *Slide 9 What has to happen for this to be done? 1) chromosomes have to duplicate themselves (CORRECTLY) 2) cell division *Slide 11 The Cell Cycle 1) Interphase G1, S, G2 2) Mitotic Phase Mitosis, Cytokinesis *Slide 12 - condensing & getting ready to divide SIDE NOTE-> Mitosis = daughter cells are identical to parent cells ( not the case in Meiosis) *Slide 13 - Chromosomes whether it’s duplicated or not ( sister chromatid) the individual when they are stuck together but the whole thing is a chromosome *Slide 14 - Prometaphase-> when things start lining up - some microtubules are for enlongating others are for sticking to the chromosomes -Mitosis -> doesn’t matter where the pairs(1&1, 2&2) are ,,,,different from Meiosis *Slide 15 - LINED UP HEAD TO TOE - metaphase, when they are pulled apart *Slide 17 - Telophase- nucleous starts forming *Slide 18 - cutting is not part of Mitosis *Slide 22 - some genetic change in a cell that makes it cancerous - cancer cells don’t listen to the check points & “stop” signals but rather keeps dividing when they shouldn’t *Slide 23 -Picture B – even though the cells are touching & surface is covered, cancer cells keep dividing *Slide 24 -Benign Tumor – cells stay where they are & surgery can take them out -Metastatic Tumor- cells spread, bad tumor to have *******Cancer cells start killing healthy cells & that’s why cancer cells are detrimental *Slide 27 - Adults – cells that are continually being replaced -Childhood – Something goes wrong during development stage (be able to tell WHY adults and children get different types of cancers) 2-4-2016 *Slide 2 - genetic variation in genes - more common to have variety *Slide 3 - in-between is most common - slow gets eaten, population slowly gets rid of slow gene *Slide 5 - Egg & sperm are very different from any other cell ( MEIOSIS) - Sperm 23 + egg 23 = 46 kid *Slide 6 - Sperm/egg -> sex cells/ haploid/ gametes/ n - All cells that aren’t sperm or egg -> somatic/ diploid/ 2n - Chromosomes -> 22 autosomes + 1 sex chromosomes = no pairs -> Miosis (cells responsible for passing genetic information) -Chromosomes -> 44 autosomes + 2 sex chomosomes (homologous pairs) = Mitosis *Slide 8  LINE UP: holding hands Meiosis: Step 1 – separate chromosomes not sister chromatids Step 2- split sister chromatids *Slide 9 1) prophase- find eachother 2) metaphase – hold hands *Slide 10 - Tetrad -> 2 homologous pairs stuck together - crossing over-> exchange DNA b/w non-sister chromatids *Slide 11 - Sister chromatids are still together *Slide 12 - functionally nothing different than Mitosis but the product is different *Slide 13 -Mitosis-> want to produce same thing - Meiosis -> cut pairs in half


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