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Chapter 14 Notes

by: Tori Morrison

Chapter 14 Notes PSY 340

Tori Morrison
Cal Poly

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These notes cover lecture and textbook information on Mental Illness
Class Notes
mental, illness
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This 2 page Class Notes was uploaded by Tori Morrison on Monday April 18, 2016. The Class Notes belongs to PSY 340 at California Polytechnic State University San Luis Obispo taught by Selby in Spring 2015. Since its upload, it has received 9 views. For similar materials see Biopsychology in Psychlogy at California Polytechnic State University San Luis Obispo.

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Date Created: 04/18/16
Chapter 14: Mental Illness concordance rate- the likelihood of the presence of a trait in a pair of twins Anxiety Disorders possible causes 1. hypersensitivity of receptors located on cells containing norepinephrine in the locus coeruleus 2. lack of GABA or receptors for GABA (bc GABA is inhibitory leads to overstimulation 3. lack of seratonin Panic disorder- frequent and unexpected panic attacks treatment- medications that increase 5HT OCD basal ganglia involved- repetitive thoughts and behaviors lack of 5HT and dopamine too active in this area treatment psychotherapy cingulotomy- brain surgery where piece of basal ganglia is removed PTSD tend to have lower levels of cortisol (born with) Schizophrenia treatments 1896- focal infection theory. psychosis caused by infections. treatment- removing various organs 1930s- Manfred Sakel insulin therapy 1935-46- Egas Moniz and Walter Freeman prefrontal lobotomies. freeman- transorbital lobotomies 1950-90- Thorazine & other meds which blocked dopamine receptors side effects blocks other receptors as well, like acetylcholine-dry mouth, blurred vision, constipation norepinephrine and histamine- sedation dopamine blockade in CNS- restlessness, agitation, motor incoordination tardive dyskinesia- late-appearing movement disorder 1990- now- clorazil blocks DA and 5HT 5HT may have something to do with psychotic symptoms and/or side effects receptor dissociation hypothesis- block DA receptors long enough to produce antipsychotic effects in mesolimbic pathway, but then disconnect before side effects are produced Affective Disorders- Disorders of mood exogenous depression- normal moods MDD Ipronazid blocked reuptake of 5HT and NE monoamine hypothesis of depression- caused by too little 5HT or NE receptor dysregulation hypothesis- also a result of too many receptors in postsyaptic membrane. treatment monoamine oxidase inhibitors- blocks monoamine oxidase from metabolizing NE and 5HT Tricyclic Antidepressants- block reuptake of NE and 5HT SSRIs- selectively block reuptake of seratonin Bipolar Disorder lithium calming effect but low safety margin


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