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Lecture notes

by: Avi Fox

Lecture notes KIN202 Wesley Smith Applied Nutrition for Health & Performance

Avi Fox
Applied Nutrition for Health and Performance
Wesley Smith

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All the lecture notes for this week
Applied Nutrition for Health and Performance
Wesley Smith
Class Notes
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This 2 page Class Notes was uploaded by Avi Fox on Friday February 27, 2015. The Class Notes belongs to KIN202 Wesley Smith Applied Nutrition for Health & Performance at University of Miami taught by Wesley Smith in Spring2015. Since its upload, it has received 114 views. For similar materials see Applied Nutrition for Health and Performance in Kinesiology at University of Miami.

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Date Created: 02/27/15
0 2 2 3 0 22 7 Role of lipoproteinscholesterol in heart disease Pathogens of heart disease Coronary Heart Disease Role of blood pressure in ammation oxidative stress and blood glucose Nutritional mechanisms of prevention 1 cause of death in the world Coronary artery disease food born illness that develops over time Could tell between Vietnamese and Americans by looking at the plaque in the arteries It starts at a young age and by 4050 have advances legions on the arteries Coronary arteries the small branches that carryoxygenated blood to the heart The plaque formed here doesn t give enough blood ow to the heart then cant give any blood to brain or the rest of the body Plaque forms behind the artery lining that causes in ammation and then it builds a bubble that can burst that causes a heart attack when a clot forms to stop it Bypass is trying to bypass these areas Insert a catheter with balloon and then insert a stent Extensive fatty deposits on the wall that can rupture 1 Damage to endothelial lining one cell layer thick smooth allows blood to ow without anything blocking it Damage can be caused by Stress from high blood pressure sugaracts as a reducing agentirritate the wall when its high in blood free radicals and in ammation 2 Creates permeable area and LDL gets in Cholesterol Lipoproteins comes out of liver as VDLDL deposits fat along body becoming LDL One enzyme created is Lipoprotein lipase removes fat and cholesterol from blood If you sit for a lot you lose a lot of those Gets to damage in the heart and gets sucked through the permeable area in the endothelial lining 3 The LDL turns rancidbeing oxidized activating in ammation recruiting 4 The causes the walls to get stickier adhesion molecules cause the walls to catch the white blood cells Macrophage big eater digests ldl creating a big foam 0611 Foam cell causes more oxidative stress Atherosclerosis legion for every 1 pts you raise HDL you reduce risk of heart disease by 8 Raise by 4pts reduce risk by 32 HDL binds to foam cell and remove the cholesterol from inside your body and out of the artery Counters LDL People with small LDL have high triglycerides and small HDL If LDL is large doesn t get in High level of triglycerides is high level of insulin Eventually plaque can rupture as it expends into vessel Platelets come in and try to stop it CAD Risk factors 1 2 gt195quot Family history firstdegree relative male before age of 55 female before age of 65 Estrogen protects arterial wall Smokers have a 300 increase risk When you smoke all the particles rush to the heart The arteries that line a smoker s heart are much stickier than the average person High blood pressure have it measured on 2 different occasions and the systolic contract top is 140 or higher and the diastolic is 90 or higher LDLgt 130mgdl or HDLlt40mgdl Elevated fasting blood glucose Obese BMIgt30 Waistgt 40 Sedentary lifestyle 150 minutes a week of brisk walking or 75 minutes of vigorous training HDL 60 or more subtracts one risk factor General process of plaque forming behind the artery wall lifestyle change is as effective or greater than medicine


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