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Chapter 4 KINE 1400 Notes - Exercise Physiology

by: Abby Chase

Chapter 4 KINE 1400 Notes - Exercise Physiology kine 1400

Marketplace > University of Texas at Arlington > Kinesiology > kine 1400 > Chapter 4 KINE 1400 Notes Exercise Physiology
Abby Chase
GPA 4.0

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About this Document

These notes will go over Chapter 4 from the Textbook Fitness Professionals Handbook (6th ed.) including notes from Dr. Keller's power points.
David M Keller
Class Notes
Chapter, 4, KINE, 1400, notes, exercise, Physiology, Keller, Fitness, professionals, handbook, University, Of, texas, AT, arlington, UTA, Kinesiology, anatomy, abby, chase
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This 4 page Class Notes was uploaded by Abby Chase on Thursday September 15, 2016. The Class Notes belongs to kine 1400 at University of Texas at Arlington taught by David M Keller in Fall 2016. Since its upload, it has received 13 views. For similar materials see INTRODUCTION TO EXERCISE SCIENCE in Kinesiology at University of Texas at Arlington.


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Date Created: 09/15/16
Chapter 4: ExercisePhysiology Energy and Work  Immediate energy sources o 1-5 seconds o ATP, PC o Anaerobic  Short term energy sources o <2 min o Glycolysis (breakdown of CHO) o Anaerobic  Long term energy sources o >2 minutes o Muscleglycogen, glucose,plasma FFA o Aerobic ATP Production  Anaerobic exercisedecreases %of ATP production with more time  Aerobic exerciseincreases %of ATP production with more time MuscleFiber Types: Genetics,Sex, Training  Distribution has a wide variation  Genetic component (fraternal twins)  Fast-twitch fibers do not convert to slow-twitch with training  Training increases oxidative capacity o Increasing mitochondrial number o Capillarydensity MuscleFiber Distribution  I (ST) – 52%  IIa (FTa) –33%  IIx (FTx) -13%  Type IIx canbe converted to IIa inresponse to aerobic training Type I (ST or SO)  Increased capillarydensity to carry O2 to mitochondria  Fatigue resistant  ATP regenerated in the mitochondria Type IIa (FOG or FTa)  Has characteristics ofboth type Iand IIx  Greater force produced than Type I o Alsoresists ffatiguedue to largenumber of mitochondria Type IIx (FG or FTb)  Primarily- anaerobic metabolism for contraction (CPor glycolysis)  Highestforce  Fatigue quickly VO2= oxygen consumption VO2inspired-VO2 expired VO2max = max rate at which oxygen can be consumed/utilized during maximal physical work Gross VO2 =Total VO2including resting requirements Net VO2 =Gross VO2-Resting VO2 Net VO2 is associatedwiththe amount of VO2being performed during exerciseminus resting oxygen uptake Maximal OxygenConsumption  VO2max measured near the end of an incremental exerciseprotocal  VO2max vs VO2peak Factors that influence on VO2max  Training – 5-25% increase(44% extreme interval training)  Heredity canaccount for up to 40 %  Gender and Age  Altitude and VO2max- inverse relationship VO2and Altitude  Altitude and VO2max – inverse relationship  Decreasen O2pressure with a risein altitude ->decreasein arterial oxygen content-> increasedHR for a given work load Carbon Monoxide  CO binds to hemoglobin and decreases O2content to blood. After 4% concentration there is a 1% drop in VO2max for every 1% CO increasein the blood Respiratory Exchange Ratio  VCO2production/ VO2consumption  RER (R)indicated fuel utilization for a given condition  Factors that influence R o Hyperventilation will increaseR value o Acute – anxiety, discomfort, pain o Chronic – metabolic acidosis o Talking and recent food ingestion Effects of diet and training on fuel utilization  HighCHO diet canincreasemuscle glycogenstores  Heavy training prior to ingesting CHP  Sports drinks maintain blood glucoseduring prolonged exercise  HR/Time/Ventilation graph 4.10  O2uptake/Time graph 4.9 Effectof Training on LactateThreshold  Graph 4.12  Anaerobic glycolysis  Measured inthe blood  Canbe used as a marker of fitness/performance Ventilatory threshold  Graph 4.13  Occurs at a higher work rate following training Variables that Determine Oxygen uptake  VO2= cardiac output x (a-V)O2difference  Cardiac output – HR and Stroke Volume Effects of Training on HR response to graded exercise  Graph 4.14  Max HR is not influenced by the amount of training  HR will be lower during exerciseduring post-training but you will still beable to reach max HR by reaching a higher grade  HR and grade are linear Cardiac Output  Graph 4.16  Stays about the sameduring pre-training and post-training  Max output is increasedfollowing training O2Extraction  Stays the samebut increases following training  Graph 4.17  BP comes from LeftVentricle Blood Pressure  Training lowers blood pressure  During exercisesystolic will raiseanddiastolic will lower Effects of detraining  Stoke volume, VO2 max, Qmax,(a-v)O2max all will decreasewith amount of days Core Temperature  Graph 4.25  Heat comes from metabolic efficiency  Heat loss –graph 4.26


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