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Review of chapter 4 & 7 before the test

by: Emma

Review of chapter 4 & 7 before the test BIOS 213

Marketplace > University of Nebraska Lincoln > BIOS 213 > Review of chapter 4 7 before the test

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Pre test review of two particular subjects
Human Physiology
Class Notes
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This 2 page Class Notes was uploaded by Emma on Monday October 17, 2016. The Class Notes belongs to BIOS 213 at University of Nebraska Lincoln taught by Bachman in Fall 2016. Since its upload, it has received 3 views.


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Date Created: 10/17/16
Review of Lecture 7 & 4 Autoregulation in the nephron-function is to maintain GFR only works when you have slight variation in blood pressure Myogenic Response- due to stretch in the afferent arteriole leads to reflexive vasoconstriction (just the response of the muscle tissue) Tubuloglomerular feedback- macula densa cells of the juxtaglomerular apparatus -Macula Densa cells monitor filtrate. Facing the lumen of the nephron (specifically Na+ concentration) -if Na+ is high it means GFR is high -release a paracrine (locally acting signal molecule) result is vasoconstriction -if Na+ is low it means GFR is low -macula densa cells stimulate granular cells to release renin Granular cells-release renin Renin-enzyme, goes into blood, converts angiotensinogen to An 1 and to Angio converting enzyme and then to angiotension 2 Angiotension 2 causes vasoconstriction especially of the efferent arteriole, GFR will increase as result Granular cells respond to macula densa cells when the MDC detect low sodium Measure BP in afferent arteriole Decrease in BP leads to an increase in renin OAT-in nephron Function to secrete xenobiotics/drugs/exogenous material usually in the form of an organic ion (mostly negatively charged) -Tertiary active transport on a cell of distal tubule 1. basolateral Na+/K+ pump & Na+ in ICF \ 2. apical Na+/dicarboxylate symporter, brings dicarbox into cell -Secondary active transport Then tertiary 3. Basolateral OAT, transporter (antiporter) that moves organic ion from plasma or ICF into the cell as the alpha keto goes down its concentration gives us increase in A- in ICF more transporter move it into the lumen AMPA-R & MMDA-R are postsynaptic receptors in synapses for ‘learning’ postsynaptic modulation -glutamate binds both, neurotransmitter -glutamate binds the AMPA-R, it opens and you get sodium influx and the cell depolarizes -Glutamate binds the MMDA receptors it opens and you get a calcium influx adds to depolarization (increases epsp) up regulation of AMPA-R


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