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by: Mrs. Adriana Beier


Mrs. Adriana Beier
GPA 3.69

Nomeli Nunez

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About this Document

Nomeli Nunez
Class Notes
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Popular in Nutrition and Food Sciences

This 16 page Class Notes was uploaded by Mrs. Adriana Beier on Sunday September 6, 2015. The Class Notes belongs to NTR 326 at University of Texas at Austin taught by Nomeli Nunez in Fall. Since its upload, it has received 56 views. For similar materials see /class/181391/ntr-326-university-of-texas-at-austin in Nutrition and Food Sciences at University of Texas at Austin.

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Date Created: 09/06/15
Nutrition and Health Nomeli R Nunez MRH PhD Department of Human Ecology Division of Nutritional Sciences University of Texas at Austin High fat diet Friar exerciser Ga natics Exercise Atherosclerosis Hypertension high blood pressure smoking diabetes obesity high cholesterol family history of heart disease and a sedentary lifestyle are involved Narrowed Plaque Emery Cutsection of artery E m U m 1 E El 2quot Ghulastam l crapnails Hanj blood cell Mamuphaga In lln cal Fa I dun nails Liver disease 7 of War damage Source DER SPIEGEL SEEM Liver Fibrosis Cirrhosis deposit m fa causes liver enlargernml Scar tissue forums Crown of autumn lit12 1155 ue destroys Ii39uer tellls The liver the largest organ in the body is essential in keeping the body functioning properly It removes or neutralizes poisons from the blood produces immune agents to control infection and remove g m bacteria from the blood It makes proteins that regulate blood clotting and produces bile to help absorb fats and fatsoluble vitamins You cannot live without a functioning liver In cirrhosis of the liver scar tissue replaces normal healthy tissue blocking the ow of blood through the organ and preventing it from working as it should Cirrhosis is the twelfth leading cause of death by disease killing about 26000 people each year WT AZlPF1 Cancers Associated with Obesity W In Men Breast postmenopausal Prostate Endometrial Stomach Cerv1cal Uterine Colorectal Ovarian Kidney Colorectal LiverGall Bladder Kldney Liver Gall Bladder Pancreatic P t39 ancrea 1c Esophageal Esol haveal Calle et a1 NEHVI 42403 The relationship between BMI and the risk of developing type 2 diabetes 100 Women 540 m 70 I Men 0 5 o 40 L 39U x N 10 0 O gt p quot6 x 5 9 m 0 lt22 lt23 23 24 25 27 29 31 33 35 239 249 269 289 309 329 349 Body mass index kgm2 Type 1 Diabetes Type 1 diabetes formery called juvenil diabetes or insulindependent diabetes is usually rst diagnosed in children teenagers or young adults In this form of diabetes the beta cells of the pancreas no longer make insulin because the body s immune system has attacked and destroyed them Treatment for type 1 diabetes includes takin insulin shots or usin an insulin pump making wise food choices exercising regularly taking aspirin daily for some and controlling blood pressure and cholesterol Type 2 Diabetes Type 2 diabetes formerly called adultonset or noninsulindependent diabetes is the most common form of diabetes People can develop type 2 diabetes at any age even during childhood This form of diabetes usually begins with insulin resistance a condition in which fat muscle and liver cells do not use insulin properly At rst the pancreas keeps up with the added demand by producing more insulin In time however it loses the ability to secrete enough insulin in response to meals Being overweight and inactive increases the chances of developing type 2 diabetes Treatment includes taking diabetes medicines making wise food choices exercising regularly taking aspirin daily for some and controlling blood pressure and cholesterol The long term effects of diabetes can be divided into 1 Macrovascular complications affect the larger blood vessels such as those supplying blood to the heart brain and legs The most common macrovascular fatal complication is coronary artery disease Strokes are also a common cause of disability and death in people with diabetes Microvascular complications affect the small blood vessels such as those supplying blood to the eyes and kidneys The microvascular complications of diabetes are retinopathy nephropathy and neuropatny Cases skyrunkal 111 quotmm dun ages 15 m 79 mwa disgmsed w Shela needy nipva 491mm m 193 m 14 mll nn M20115 4 W The fight against diabetes 311 5 may a 11m 154 Glucose clearance from blood Intestine RBC Glucose Pancreas or Fat Liver Fatty acids insulin 0 1 7 lt f fmv 7 quot 1quot m m quotj x 2 V 94144 hum ELL UL MKLE UJAJZKL 3 il Vii Plasma w 39 w 3 glumsa GLLUT Glucase s Glucazsew pmspham O Glucose Body Weights grams Diet induced obesity Obese Overweight Lean 6 8 10 12 14 Weeks 0n dlets iii i 161820 Obesity induces insulin Resistance T00 m h insulin 39gt Insulin o 39l quot r 39 39 ha E Carry399 39 air we Iii glucose 1 3 1 g Glucose GlucoseSphosphat Body weight and blood glucose Clearance 500 400 glucose mgd1 Inject glucose


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