Week Five with Pope
Week Five with Pope ANISCI 3140
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This 4 page Class Notes was uploaded by JessAnn22 on Sunday September 27, 2015. The Class Notes belongs to ANISCI 3140 at Ohio State University taught by Pope in Fall 2015. Since its upload, it has received 36 views. For similar materials see Animal Physiology in Animal Science and Zoology at Ohio State University.
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Date Created: 09/27/15
Week Five with Pope QUIZ on wednesday Ql Another name for a Schwann Cell Al Neurolemmocyte Q2 The ventral branch should NOT contain A2 Vagus Neurons Red Blood Cells RBCs 23 water 13 Hemoglobin Hb ea RBC 280 mill Hb molecules Heme groups with iron that will bind oxygen Globins change their shape because of pH relaxed pick up oxygen in lungs release oxygen in tissues purpose of Hb is to carry the oxygen Anemia most common blood disorder low amount of HbFe Can lead to hypoxia low 02 to tissue Ischemia low blood ow because of an obstruction Sickle Cell Anemia low QUALITY of Hb mutation of 1 amino acide in beta globin hemoglobin molecules bind and distort to become sickle cell in shape Begins destroying RBCs genetic homozygous recessive Thalassemia low QUANTITY of Hb too few globins synthesized Anemia in Lambs White conjuctivia parasitic infection Erythropoiesis making of RBCs Erythropoietin HORMONE that stimulates production of RBCs EPO erythropoeitin hematopoietin hemopoietin Kidneys detect reduced Oglearming eapacity oi blood Reduced oxygen carrying capacity When less 02 is delivered to the kidneys they secrete elrylhropoletin into blood Developing erythrocytes in red bone marrow a Erythropoietin stimulates eryih ropoiesie He l39eves by bone marrow E Additional circulating erythrocytes increase Qgcarrying capacity of blood 23 Increased oxygen cerwing capacity increased Og carry ilng capacity relieves initial stimulus that triggered erythropoietln secretion Erythrocytes 6 FIGURE Ii3 Control of erythropoiesis Diagram of erythropoeisis Making RBCs takes 46 days Early in the embryo YOLKSAC makes RBC then it shifts over to the LIVER then BONES in adults Baby Pigs Anemia newborns average 3 lbs at 21 days they are 14 lbs 47x bigger in 3 weeks such fast growth they cant make blood fast enough Afb V Alb sex differentation Do not mentally sexually differentiate until 3 weeks of age Fetal Hb fetus lives in a HYPOXIC ENVIRONMENT Fetal Hb Has 2 alpha globins and 2 gamma globins Shortly after birth there is a rush of BILIRUBIN Hb breakdown Heme breakdown Can cause liver damage blood disease block bile duct wave of fetal Hb breakdown alpha and gamma no beta heme broken down in liver to bilirubin gives yellow color Body gets rid of it through poop Babies who cant break it down right away jaundice Exposure to UV helps break it down Fluid portion of the blood is PLASMA PLASMA v SERUM plasma not clotted serum clotted HORSES AND PIGS not uncommon to get SERUM CLOTS serum plug clotting factors in serum not all got down into the clot Plasma 92 water 8 plasma protein 2 ions solution urea creatine waste Plasma Proteins 60 ALBUMIN from liver Low molecular weight but high abundance helps maintain osmotic pressure weakly bind carry waste garbage bucket of blood 4 FIBRINOGEN forms strong clots 1st to quick platelet plug Then brin plus RBCs plus more platelets blood clot 36 GLOBULINS immunoglobulins antibodies increases when ghting an antigen Electrophoretic patterns can be a diagnostic tool seeing if they are making antibodies high gamma reading Capillary Fluid Dynamics microcirculation diagram of cap bed interstitial extracellular uid surrounds capillaries Drop in BP is key when blood travels through caps because of increase in surface area 2 major factors 1 hydrostatic pressure BP pressure inside the lumen pushing against walls Drops in caps 2 osmotic pressure pp concentration in blood albumin helps control it Keeping albumin wanting to go across membrane Normal Flow 32 HP 285 2 opposing OP 3223 9 FILTRATION PRESSURE uid being pushed out because HP gt OP 39d39 EnCiJa r EhLE39il I ELEGIIFFHquotE1 39 kph 15 r 1 739 mint EE hi U q39j tl hf ihal rm 95 asrcra rm a semis f jg Ephir rcberi Iiipen l mr ihi39mgh Ema 512 mm Eq a r i i m 51 in lgtili pmm lm lu dmb m quottram EJ39EIDJE39I39E I39IT 39inrleHTEiz quotMTImml tu hl EhEHFHEH Irtj mam9 Iruca m iJ UEHrELm lFligEmE 11 Mammy HP in p l w ARTERIOLE END 152851523 8 REABSORPTION PRESSURE uid going into caps because OP gt HP