Notes for 10/22
Notes for 10/22 BIOL 3301 - 002
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BIOL 3301 - 002
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This 1 page Class Notes was uploaded by Ashley Lutz on Monday October 26, 2015. The Class Notes belongs to BIOL 3301 - 002 at University of Texas at Arlington taught by Laura D Mydlarz in Summer 2015. Since its upload, it has received 23 views. For similar materials see CELL PHYSIOLOGY in Biology at University of Texas at Arlington.
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Date Created: 10/26/15
102215 Bulk transport topics Clatherin coating is the coating for bulk transport vesicles both coming into the cell and leaving the cell Exocytosis what the cell spits out Signalmediatedregulated secretion is used for things like neurotransmitters that are only released at certain times in response to a specific stimuli The vesicles will be altered till they are tight packets with almost nothing but the things to be secreted The vesicles are coated in proteins and other non phospholipid components so that the phospholipid bilayer is almost overrun with them They39re not simple balls filled with stuff Endocytosis is used for gaining the nutrients the cell needs stopping the effects of different stimuli horomones etc and to destroy toxicdangerous things such as bacteria and toxins This is for the larger things that can39t fit through a portion of the lipid bilayer Phagocytosis is the mega version of endocytosis The early endosome is acidic in starts the breakdown of different things this eventually changes from early to late endosome and late endosome to lysosome where everything is finally fully digested Receptor mediated endocytosis requires receptors to stimulate the cell to form a clatherin coat around the vesicle that forms as the different ligands bind it forces the lipid bilayer to change shape around the molecule The clatherin coat also helps this shape to form The rest of this process follows what we looked at for all the other transport vesicles Cholesterol is carried by LDL39s in the blood LDL39s move to the cells HDL39s move to the liver High LDL the LDL gets between the cells in the arteries the artery initiates in ammatory immune response which sticks the LDL39s in even more then everything starts sticking together with all the fibrous tissue that is forming to try to heal HDL cannot help at this point Proteasome breaks up misfolded old proteins in the cytosol or the ones that are stuck in the ER The backdoor to getting ER proteins out if they are stuck misfolded etc The Proteasome is made of a group of enzymes that pull the protein through itself and break down each part it is pulled through in smaller than tertiary form Proteasomes can only break down tagged proteins This protein is only 76 amino acids long and is added to the proteins that are faulty It is found in almost all organisms with proteasomes Ubiquitin the kiss of death molecule is added en mass to the proteins that are to be shredded
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