Energy Metabolism KIN 585
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This 3 page Class Notes was uploaded by Edwardo Schultz on Friday October 30, 2015. The Class Notes belongs to KIN 585 at University of Massachusetts taught by Staff in Fall. Since its upload, it has received 10 views. For similar materials see /class/232212/kin-585-university-of-massachusetts in Kinesiology at University of Massachusetts.
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Date Created: 10/30/15
Lecture 3 Questions 1 What are 2 ways to change the AG in a reaction 2 Using the equation AGAG RT Ln productsreactant If AG 5 how would you make AG negative What would you do to the ratio of products to reactants 3 Why does coupling the reaction Glucose Pi gt Glucose6 P with ATP gt ADP Pi make it a more favorable reaction more likely to go to completion 4 Reactions such as glucose b pyruvate overall have a AG However within the pathway there are reactions that have very positive AG s Explain how this reaction is able to go to completion Km 585 Energy Metabolism Spring 2009 Lecture 29 Insulin Resistance Insulin Receptor Glucose i e N w t 391 Fatty Acyl CollV quot lll Malonyl CoA 9 Glucose Acelyl CoA 7K mmmm a imiriismi in szvnn KeW and Mandamo ma 1 How does insulin resistance alterthe normal actions of insulin on a skeletal muscle b the livel c adipose tissue Kin 585 Energy Metabolism Spring 2009 2 Briefly how does the quotglucosefatty acid cyclequot explain the connection between elevated plamsa fatty acids and decreased blood glucose uptake in obese humans 3 What piece of evidence argues against the quotglucosefatty acid cyclequot as a mechanism underlying insulin resistance 4 Briefly how do the new models explain the connection between fatty acid oxidation versus storage and insulin resistance 5 What is the paradox that casts some doubt on the idea that high levels of stored intrmuscular lipid causes insulin resistance
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