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This 0 page Class Notes was uploaded by Kenya Bernier I on Sunday November 1, 2015. The Class Notes belongs to MBI111 at Miami University taught by JohnStevenson in Fall. Since its upload, it has received 12 views. For similar materials see /class/233344/mbi111-miami-university in Microbiology at Miami University.
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Date Created: 11/01/15
Microbiology EXAM 2 STUFF Feb 18 2010 Diseases transmitted by skin infections Epidermis Physical barrier High concentration of microbial bi products Opportunistic microbiota Epidemiology of Skin Dieseases Caused by normal microbiota or exogenous microbes introduced via Cuts abrasions burns or bites of animals or arthropods Moisture Corticosteroids Triggers immunodeficiency Acne propionibacterium acnes Inflamation pus buildup Phagocyte build up they get killed pus is the guts of phagocytes Caused when epidermal cells and sebum work their way to the skin surface and can cause pores to clog People with influxed hormones Acne Treatment RetinA causes dry skin What I use Accutane Terrotogen with lots of side effects espec Birth lots of restrictions on it CTAs Oral contraceptives female Photodynamic therapy light Multiple treatments Acne Prevention Decrease sebum accumulation by personal hygine Depress bacterial growth with topical benz peroxide containing meds A good diet isn t prevention Acne is controlled compared to these next few Impetigo scientific name Abscess based disease BIGGER and will tend to s p r e a cl Staphylococcus aureus andor stetococcus pyogenes Thinwalled abscesses that look like blisters or weeping lesions w scabs Blood poisoning possible Impetigo Prevention and Treatment Prevention Personal Hygine Avoid contact with leasions on others Treatment Topical Meds Furuncle boil MRSA Infected skin follicle leads to formation of an abseess Carbuncles clusters of furuncles Necrotic infection causing death of skin cells Blood poisoning toxemia possible due to systematic distribution of toxin bacteria Prevention of Furuncle Personal hygine Avoid contact with other individuals who may have it This becomes a problems in institutions where people don t leave jail Necrotizing Fasciitis streptococcus pyogenes About 20 fatal cases Rapidlyspreading infection leads to destruction of the tissues flesh eating disease Starts out as ceuitis Hemmorhaging in the dermis Abscesses throughout body eventually open up Skin rips due to swellng and pressure Toxemia Treatment Prevention Treatment Sometimes skin grafts to fix tears Debridement Removing dead and dying tissue Remains antibiotic succeptible Not much in the way of prevention Candidiasis candida albicans Opportunisitic Dimorphic fungus is a member of the normal microbiota Enhanced by chronic skin moisture espec In immunosuppressed individuals AIDS diabetes etc Grows on skin and induces brignt red inflamation blisterlike superficial lesions or more severe lesions Related to hypersensitivity Candidiasis Complications Systematic infection It can lead to fulminating disease and death Treatment of Candiasis Creams and oral meds Prevention Avoid excessive skin moisture 22310 Tineas Ringworm Dermatophytic fungi such as Epidermophyton Prevalent in athletes Frank upfront lttineas Pathogenesis Superficial skin infection causes granulomas on skin Headscalp tinea capitis Jock itch granulomas on groin tinea cruris Skin tinea corporus Foot tinea pedis Treatment of Tineas ringworm Lamisil Topical Lotrimin Shampoo with selenium Griseofulvin Oral Prevention of Ringworm Avoid contact with one who has it Sporotrichosis Spores of this fungus in soil are transmitted to humans MORE ON MAIN PPT
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